Hyperactive mariner transposons are created by mutations that disrupt allosterism and increase the rate of transposon end synapsis
(2013)
Journal Article
New applications for transposons in vertebrate genetics have spurred efforts to develop hyperactive variants. Typically, a genetic screen is used to identify several hyperactive point mutations, which are then incorporated in a single transposase gen... Read More about Hyperactive mariner transposons are created by mutations that disrupt allosterism and increase the rate of transposon end synapsis.